The Neurometabolic Cascade of Concussion.
By C. Giza, David A. Hovda. Journal of athletic training, 2001
1,424 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/09b6cf20644255c32b7f444ef45e559118e2e534
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Citations in the corpus
- The New Neurometabolic Cascade of Concussion (2014, unknown): “The basic neurobiology of concussion/mild TBI has been elucidated in animal models, is increasingly corroborated in human studies, and has been described as a neurometabolic cascade of events that involves bioenergetic challenges, cytoskeletal and axonal alterations, impairments in neurotransmission”
- American Medical Society for Sports Medicine position statement: concussion in sport (2012, background): “Developmentally younger brains have less established engrams and may have less cognitive reserve than more mature brains.(9,64) This may account for the demonstrated increase in time to recovery from concussion seen in younger athletes.”
- Acute effects and recovery time following concussion in collegiate football players: the NCAA Concussion Study. (2003, background): “Studying the course of recovery of postconcussive abnormalities is a critical step toward determining the interval during which a concussed brain may be most vulnerable to reinjury and establishing evidence-based guidelines for safe return to play by athletes after concussion.(2) The purpose of this”
- Cumulative effects associated with recurrent concussion in collegiate football players: the NCAA Concussion Study. (2003, background): “Although this theory has yet to be confirmed in a human model, animal research has identified acute metabolic dysfunction following cerebral concussion that might explain the increased neuronal vulnerability that can exist for several days following injury.(26-30) The purposes of this study were to ”
- Chronic Traumatic Encephalopathy: A Potential Late Effect of Sport-Related Concussive and Subconcussive Head Trauma (2011, background): “and cytoskeletal disturbances, which trigger the pathologic cascade that leads to CTE in susceptible individuals.(15,16) The onset of CTE is often in midlife, usually after athletes have retired from their sport.”