Interactions of the Drosophila gap gene giant with maternal and zygotic pattern-forming genes.

By Elizabeth D. Eldon, Vincenzo Pirrotta. 1991

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Negative Regulation of Active Zone Assembly by a Newly Identified SR Protein Kinase (2009, background): “We, therefore, examined mutations in genes that could represent SRPK79D-dependent negative regulators of translation, such as aret (bru), cup, pum, nos, and sqd [38–44], reasoning that the loss of such a translational inhibitor might result in the ectopic synthesis of AZ proteins, ultimately leading”
Control of Drosophila body pattern by the hunchback morphogen gradient. (1992, background): “Localized Kr, kni, and gt transcription is first observed during nuclear cycles 11 and 12 (Knipple et al., 1985; Nauber et al., 1988; Eldon and Pirrotta, 1991; Kraut and Levine, 1991a), arguing that graded hb protein is active at this time, if not earlier.”
The giant gene of Drosophila encodes a b-ZIP DNA-binding protein that regulates the expression of other segmentation gap genes. (1992, methodology): “This is consistent with its nuclear localization and with the effects of giant mutations on the expression of other gap genes (Eldon and Pirrotta, 1991; Kraut and Levine, 1991a,b; Small et al., 1991).”
Autonomous concentration-dependent activation and repression of Krüppel by hunchback in the Drosophila embryo. (1994, background): “seen in F are typical for the gt phenotype (Kraut and Levine, 1991; Eldon and Pirrotta, 1991).”