Mouse models in non‐alcoholic fatty liver disease and steatohepatitis research

By Q. Anstee, Robert D. Goldin. International journal of experimental pathology (Print), 2006

750 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/086f33cd5136a94a2093da6bae7b279d8cedca01

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In Endocrinology, Diabetes and Metabolism

1. M. Pinzani

In Epidemiology

1. M. Pinzani
2. Patrice D Cani

Citations in the corpus

• The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome (2010, background): “Increased influx of free fatty acids Free fatty acids that result from lipolysis of subcutaneous or visceral adipose tissue depots circulate to the liver, and may contribute to intrahepatic lipid accumulation”
• Increased intestinal permeability in obese mice: new evidence in the pathogenesis of nonalcoholic steatohepatitis. (2007, methodology): “In this study, we used two animal strains, ob/ob and db/db mice, which have been extensively used as models of obesity (3).”