Myelin-associated glycoprotein interacts with the Nogo66 receptor to inhibit neurite outgrowth.

By M. Domeniconi, Zixuan Cao, Tim Spencer, Rajeev Sivasankaran, Kevin C. Wang, E. Nikulina, Noriko Kimura, Hong Cai, Kangwen Deng, Ying Gao and 2 more. Neuron, 2002

602 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Developmental Neuroscience

View Paper: https://www.semanticscholar.org/paper/083a092657e00eaf3a7c49d662dc2b7b1c3bd848

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. J. Gensel
2. K. Nave
3. B. Trapp
4. G. Ferraro
5. L. Mckerracher
6. A. Fournier
7. David J. Loane
8. Alok Kumar

In Developmental Neuroscience

1. K. Nave
2. G. Ferraro
3. L. Mckerracher
4. B. Trapp
5. A. Fournier

Citations in the corpus

• Identification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cord (2009, methodology): “M2 macrophages overcome axon growth inhibition by CSPG and myelin CSPG and MAG are potent inhibitors of axon growth (Canning et al., 1996; Domeniconi et al., 2002).”
• Rho signaling and axon regeneration. (2012, background): “All three growth inhibitory proteins share the ability to bind to the same receptors, the Nogo receptor 1 (NgR1) (Domeniconi et al., 2002; Fournier, GrandPre, & Strittmatter, 2001; Wang, Koprivica, et al., 2002) and the paired immunoglobulin-like receptor (PirB) (Atwal et al., 2008).”
• Axon-glial signaling and the glial support of axon function. (2008, background): “Instead, MAG’s role as one of several myelin-associated and Nogo receptor-dependent inhibitors of axonal regeneration has gained much attention (e.g., Domeniconi et al. 2002).”
• The role of the Rho GTPases in neuronal development. (2005, background): “A number of growth inhibitory molecules involved are derived from myelin and include myelin-associated glycoprotein (MAG), Nogo-A, chondroitin sulfate proteoglycans (CSPGs), and oligodendrocyte myelin glycoprotein (OMgp) (Niederost et al. 1999; McGee and Strittmatter 2003; Yiu and He 2003; Sandvig e”