TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease

By J. Gross, J. Johnston, S. Mudri, Rachel Enselman, S. Dillon, K. Madden, Wenfeng Xu, J. Parrish-Novak, D. Foster, C. Lofton-Day and 9 more. Nature, 2000

1,264 citations recorded by the source corpus.

Subfields: Immunology, Radiology, Nuclear Medicine and Imaging

View Paper: https://www.semanticscholar.org/paper/047e27af8f3c5c1c2e9b1da5e5b5bcaf874a10ae

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In Immunology

1. F. Mackay
2. J. Tschopp
3. C. Mackay
4. R. Noelle
5. Ling-Li Lin
6. Rafi Ahmed
7. Shizuo Akira
8. Fabio Martinon

Citations in the corpus

• Association of BAFF/BLyS overexpression and altered B cell differentiation with Sjögren's syndrome. (2002, background): “In one model of BAFF Tg mice that used a lymphoid-specific promoter, the B-1 B cell compartment was enlarged (29).”
• Baff Mediates Survival of Peripheral Immature B Lymphocytes (2000, background): “Supporting this model, BAFF does not promote survival of naive mature B cells, in vitro, despite expression of both BAFF receptors on these cells (18, 22, 23).”
• Cutting Edge: A Role for B Lymphocyte Stimulator in Systemic Lupus Erythematosus1 (2001, background): “ease severity and prolong the survival in animal lupus models (8).”
• NF-κB1 p50 Is Required for BLyS Attenuation of Apoptosis but Dispensable for Processing of NF-κB2 p100 to p52 in Quiescent Mature B Cells1 (2003, background): “The expression of BLyS receptors is largely restricted to B lineage cells (7, 12), with the exception of a subset of T cells (13).”
• Murine Models of Systemic Lupus Erythematosus (2011, background): “Additionally, nonautoimmune transgenic mice overexpressing BAFF showed lupus-like symptoms [130].”