Podocyte depletion causes glomerulosclerosis: diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene.

By B. Wharram, M. Goyal, J. Wiggins, S. Sanden, S. Hussain, W. Filipiak, T. Saunders, R. Dysko, K. Kohno, L. Holzman and 1 more. Journal of the American Society of Nephrology, 2005

756 citations recorded by the source corpus.

Subfields: Nephrology, Surgery

View Paper: https://www.semanticscholar.org/paper/041e4c478f8e7c43f337dd19799da729c5def6d2

Citations in the corpus

• Molecular mechanisms of diabetic kidney disease. (2014, neutral): “Greater than 20% podocyte loss in animal models causes irreversible glomerular damage, manifesting as albuminuria; glomerulosclerosis then progresses to tubulointerstitial fibrosis and end-stage kidney failure (43, 64, 65).”