Recurrent fusion of MYB and NFIB transcription factor genes in carcinomas of the breast and head and neck

By M. Persson, Ywonne Andrén, J. Mark, H. Horlings, Fredrik Persson, G. Stenman. Proceedings of the National Academy of Sciences of the United States of America, 2009

785 citations recorded by the source corpus.

Subfields: Molecular Biology, Pathology and Forensic Medicine

View Paper: https://www.semanticscholar.org/paper/0121e4b3d9b4d324ad1008728d021c284221d7e9

Citations in the corpus

• Current landscape and future directions of therapeutic approaches for adenoid cystic carcinoma of the salivary glands (Review) (2025, unknown): “…NOTCH2 or loss of the CDKN2A/CDKN2B locus, may be involved in ACC pathogenesis, with a recurrent t(6;9) (q23;p23) translocation resulting in a fusion between the MYB proto‑oncogene, transcription factor (MYB) and nuclear factor I/B (NFIB) genes, considered to be a genetic hallmark of ACC (26).”