MicroRNA-192 in diabetic kidney glomeruli and its function in TGF-β-induced collagen expression via inhibition of E-box repressors

By Mitsuo Kato, Jane Zhang, Mei Wang, L. Lanting, Hang Yuan, John J. Rossi, R. Natarajan. Proceedings of the National Academy of Sciences of the United States of America, 2007

729 citations recorded by the source corpus.

Subfields: Cancer Research, Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/00d52d87d27a0d46748d95e12b56dd131975496b

How researchers see this paper

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In Cancer Research

1. Danish H Sayed
2. M. Abdellatif

In Molecular Biology

1. Danish H Sayed
2. H. Lan
3. Jun Li
4. Xiao-ming Meng
5. M. Abdellatif
6. P. Tang

Citations in the corpus

• TGF-β/Smad signaling in renal fibrosis (2015, background): “It is reported that miR-192 is elevated in fibrotic mouse models and TGF-β1-treated murine cells (Kato et al., 2007; Chung et al., 2010a; Putta et al., 2012).”
• MicroRNAs in development and disease. (2011, background): “On the other hand, TGF- plays a major role in the production of collagen in the diabetic kidney through inhibiting the expression of the E-box repressors, ZEB1 and ZEB2, which potentially suppress E-box-dependent expression of collagen (290).”