Airway epithelial gene expression in the diagnostic evaluation of smokers with suspect lung cancer
By A. Spira, J. Beane, Vishal Shah, K. Steiling, Gang Liu, F. Schembri, Sean Gilman, Yves-Martine Dumas, Paul Calner, Paola Sebastiani and 8 more. 2007
562 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology, Pulmonary and Respiratory Medicine
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cancer Research
In Molecular Biology
- Daniela M. Witten
- Peter J. Danaher
- Reem Nooreldeen
- Noah R. Simon
- Alice X. Zheng
- J. Rock
- Pei Wang
- H. Bach
In Pulmonary and Respiratory Medicine
Citations in the corpus
- Fused Lasso Additive Model (2014, background): “Lung S [Spira et al., 2007]: 22,283 gene expression measurements from large airway epithelial cells sampled from 97 smokers with lung cancer and 90 smokers without lung cancer, available from GEO at accession number GDS2771.”
- Current and Future Development in Lung Cancer Diagnosis (2021, background): “Further studies using these markers are necessary to apply to populations with increased risk, such as smokers without lung cancer and survivors of previous cancer [66].”
- Gradient boosted feature selection (2014, methodology): “There- SMK-CAN-187 fore, we limit the number of trees to 100 and the maximum number of instances per leaf node to 500.”
- The joint graphical lasso for inverse covariance estimation across multiple classes (2011, methodology): “roarray data We applied FGL to a dataset containing 22283 microarray-derived gene expression measurements from large airway epithelial cells sampled from 97 patients with lung cancer and 90 controls (Spira et al. 2007). The data are publicly available from the Gene Expression Omnibus (Barrett et al.”
- Airway basal stem cells: a perspective on their roles in epithelial homeostasis and remodeling (2010, background): “Furthermore, choosing ‘normal’ control samples could be complicated if predisposing genetic lesions are widely distributed in the airway and oral epithelium (Slaughter et al., 1953; Spira et al., 2007).”